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Antioxidant therapy in schizophrenia Three groups of treated and untreated patients with schizophrenia and healthy controls were analyzed for the content of antioxidant enzymes and low molecular weight antioxidants

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KYNA acts as a non-selective competitive antagonist at the glycine B site of the ionotropic N-methyl-D-aspartate receptor (NMDAR) [27, 28]

S2CID 20796904

Histological analysis revealed that Dihexa treatment increased neuronal cell counts and enhanced expression of synaptophysin (SYP), a presynaptic protein marker indicating synaptic density, in the hippocampus of APP/PS1 mice