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Additionally, glutamine-derived metabolites influence epigenetic modifications, modulating gene expression patterns associated with CSC maintenance and treatment resistance

doi:10.1016/j.psyneuen.2013.02.004

Potts EN

Therefore, depleting GSH to modulate ROS level holds significant promise as an effective therapeutic strategy for treating AML [9, 22, 23]

Similar content being viewed by others Introduction Tyrosinemia type I (hepatorenal tyrosinemia, HT-1) is an autosomal recessive condition (OMIM 276700) resulting in hepatic failure with comorbidities involving the renal and neurologic systems (for a comprehensive review, see Mitchell et al