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Results Neuronal GCLC knockout leads to brain atrophy accompanied by neuronal cell death To examine how the declines in GCLC and glutathione might affect brain homeostasis, we prepared GCLC floxed X CaMKII-Cre (GCLC-KO) mice whose GCLC expression is deleted in calcium/calmodulin-dependent protein kinase II (CaMKII)-positive neurons

The regression slope of 0.9949, being exceptionally close to unity (1.0), along with a minimal y-intercept of 1.7722, collectively demonstrates an almost perfect agreement and negligible systematic bias between these two assays