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Published research describes suppression of NF-kappaB signaling, reduced production of pro-inflammatory cytokines (TNF-alpha, IL-1beta, IL-6), and modulation of the nitric oxide system (Catania et al., 2004)

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In addition to the inhibition of arachidonic acid metabolism by COX inhibition, other possible mechanisms for the multiple effects of ASA include increased ROS production, alterations in mitochondrial function, cell signaling and induction of apoptosis [7], [19], [30], [31], [32], [33]