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Ferroptosis is strikingly driven by worsening redox imbalance, which generates and maintains high levels of hydroperoxy-phospholipids (7, 24)

In CM, more than 90% of mutations are present on the V600 codon, whereas in MM only 63% are V600 mutations, with the remainder (37%) on the G469, D594, and K601 codons (91, 93)

Furthermore, the validity of TMA/TMAO ratio as a better CVD biomarker needs to be investigated

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This binding can induce conformational changes in -helices, which in turn regulates lipid homeostasis