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* Correspondence: Yiming Zeng, [email protected] These authors have contributed equally to this work Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Furthermore, a study revealed that tunicamycin administration in the ER caused a significant increase in cell death exclusively in wild-type mice and that administering antioxidants prior to angiotensin II prevented ER stress and apoptosis in cultured cardiac cells

To our knowledge, this is the first report of a single microbial strain capable of producing TMA from one of the metabolites of the l-carnitine pathway under anaerobic conditions

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