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These changes can cause oxidative modifications to reactive thiols of structurally- or functionally-critical proteins, such as transcription factors 43 , signal transduction molecules 44 or metabolic enzymes 45 , and thereby evoke cellular defense mechanisms against oxidative damage 46

Regulation of ferroptosis can be achieved generally by interfering with iron metabolism and ROS metabolism, and the ferroptosis process can be suppressed by iron chelators, lipophilic antioxidants, lipid peroxidation, and the depletion of polyunsaturated fatty acids and correlates with the accumulation of lipid-peroxidation markers (Stockwell et al., 2017)

2005 Jun 30;1741(1-2):65-74