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These drugs inhibit cell cycle progression at G1/S checkpoint and exhibit significant improvement in progression-free survival and overall survival (in some cases) when combined with endocrine therapy (218)

Schneich et al, 1989), would directly oxidize low-density lipoprotein (LDL) (Heinecke et al, 1987

This substrate specificity of arginase for arginine (not citrulline) allows oral L-citrulline to bypass significant intestinal metabolism, entering circulation intact before primary conversion to L-arginine occurs systemically, particularly in the kidneys [41,46]

However, the mechanisms underlying the synaptic and mitochondrial aberrations in FXS have yet to be established

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