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BRB disruption is a primary pathogenic mechanism of DR and the key factor driving DR progression from non-proliferative DR (NPDR) to proliferative DR (PDR)

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After 18 h BSO and cysteine incubation, excess media was removed, replaced with 100 L A6 media containing 0.6 MOI frog virus 3 (FV3) (37), and incubated at 30C for 1 hour

doi: 10.2147/DDDT.S556231, Summary Keywords fibrosis, glucagon-like peptide-1 receptor agonists, inflammation, metabolic dysfunction-associated steatohepatitis, metabolic dysfunction-associated steatotic liver disease Citation Lu X and Yang L (2026) Glucagon-like peptide-1 and dual/triple receptor agonists in the treatment of metabolic dysfunction-associated steatotic liver disease: advances in mechanistic research

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