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A reliable method for producing stress ulcers in rats

In sum, it is worthy to note that lipid peroxidation (LPO) products such as 4-hydroxynonenal (4-HNE) and malondialdehyde (MDA) arise as secondary messengers of oxidative stress, which is often counterbalanced by the cellular antioxidant defense system [133]

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Furthermore, standardized tDCS parameters may inadequately accommodate the altered electric field distribution resulting from structural brain changes (e.g., cortical atrophy) in this population