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Although it serves as an essential cofactor for critical enzymes governing antioxidant defense, energy production, and structural integrity, its dysregulationmanifesting as either deficiency or excessis a common pathogenic thread across diverse cardiovascular conditions

89 Importantly, restoration of hypothalamic autophagy via nutritional interventions such as docosahexaenoic acid-acylated astaxanthin monoester (AST-DHA) supplementation or genetic activation of Atg5 ameliorates obesity and improves glucose metabolism in experimental models, 90 demonstrating that hypothalamic autophagy is not merely correlative but mechanistically regulatory

The depressurization of the NP causes a load shift from the NP to the AF under compressive loads

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