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The impact of changed redoxin function on ferroptosis could also be based on secondary effects, e.g., the increase in superoxide formation upon loss of mitochondrial redoxins [90,91] or activation of the nuclear factor erythroid 2-related factor 2 (Nrf2) pathway [92,93] , a major regulator of transcription of antioxidant and anti-ferroptotic enzymes [65]

On the other hand, the gdh3 / gdh3 mutant showed similar growth kinetics to the wild-type cells when grown on arginine as the sole carbon and nitrogen source, but the gdh2 / gdh2 strain showed minimal growth (0.009 h 1 ) and biomass yield (0.016 g L 1 ) compared to the wild-type strain (growth rate = 0.068 h 1 , biomass = 0.50 g L 1 )

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