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However, APR-246 also has p53-independent function via MQ binding to thioredoxin reductase and glutathione, leading to depletion of glutathione and accumulation of reactive oxygen species (ROS), which can feed forward p53 activation (Figure 1).1918 Indeed, the authors also show synergy in TP53 knockout mutant cell lines where there is absence of p53, albeit with less synergy than in the missense mutant model

Role of the Keap1/Nrf2 pathway in neurodegenerative diseases

Patients should be aware that some products sold as semaglutide may not contain the same active ingredient as FDA-approved semaglutide products and may be the salt formulations

He XH, Li WT, Peng WJ, Li GD, Wang SP, Xu LC