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Thus, during fibrillogenesis, v3 integrin maintains the FAs, while the contractile forces of the actomyosin fibers drive 51 integrin translocation into fibrillar adhesions
I compiled my list after doing lots of research on-line (too much time on my hands and too much to worry about) and by talking to some colleagues
Secondary urea cycle defects (intramitochondrial CoA depletion and cellular injury may exacerbate the disease)
400 The underlying neuroprotective mechanisms involve increased levels of miR-21, miR-27a, and miR-146a, along with decreased expression of target proteins
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