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[2] BPC157s interactions with the NO system have been proposed as a central mechanistic contributor to its gastrointestinal cytoprotective profile, with research suggesting potential upregulation of eNOS expression and modulation of NO bioavailability within gastric mucosal microcirculation

When GIPRAs and GLP-1RAs were co-administered to obese mice, the combination significantly enhanced both appetite suppression and weight loss compared to monotherapy [33, 34]

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Since PS-expressing eryptotic erythrocytes are cleared by efferocytosis, this mechanism might elicit immunogenic effects as well