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Considering its role as a matricellular protein, further investigation is needed to elucidate whether periostin participates in the regulation of disc cellmatrix interactions (111)

GPX4-independent antioxidant mechanisms Following the inactivation of GPX4, some cancer cells still remain resistant to ferroptosis [67, 68], implying that there are other ferroptosis defence mechanisms

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That transition is appropriate: the molecular logic is sound, the therapeutic precedents are encouraging, and the unmet need, extending not just lifespan but healthspan, the years lived in full function and vitality, is among the most significant in modern medicine