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Firstly, given the evidence that glutathione and glutamate levels in the brain are affected by a risk variant of the gene coding for the glutathione synthesis enzyme, the role of genetic influences should be investigated [22]

Accumulation of saposin in dystrophic neurites is linked to impaired lysosomal functions in Alzheimer's disease brains

This occurs because appetite returns to pre-treatment levels and the body's metabolic adaptations to weight loss persist, making weight maintenance challenging without continued intervention