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These alterations not only extend its duration of action but also enable IGF-1 LR3 to circumvent binding with IGF-binding proteins, which typically impede the biological availability and efficacy of IGF-1

Hi all, I apologise in advance, as this has probably been asked many times before

Excessive prolonged stimulation may induce steady-state downregulation of synaptic plasticity (Hurley and Machado, 2017)