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Ocular hypertension triggers the production of pro-inflammatory cytokines by immune cells, which in turn drives the transformation of neuroprotective astrocytes into a neurotoxic A1-reactive phenotype, ultimately leading to RGC degeneration [45]

As a modified peptide analog rather than a full receptor-active growth hormone construct, AOD 9604 is distinguished by its selective interaction with metabolic signaling pathways associated with lipid mobilization rather than canonical GH receptor activation

demonstrated that L-carnitine supplementation led to elevated myocardial creatine content and enhanced cardiac performance in patients undergoing CABG (Pastoris et al

, Knudsen, L

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