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Furthermore, enhancing O-GlcNAcylation through GlcN injection ameliorated PD pathogenesis induced by 6-OHDA in mouse brains, with attenuated neuroinflammation and mitochondrial dysfunction and mitigated motor deficits [188]

These patients also exhibit polymorphisms to the serotonin receptor 2A region of chromosome 13 and the human leukocyte antigen region of chromosome 6, as well as abnormalities in catechol- O -methyltransferase