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Therefore, we hypothesized that the excessive expression of GJs post-APAP exposure in hepsin / mice could be a key factor contributing to the increased severity of liver damage observed following APAP administration

It functions as a highly specific inhibitor of an enzyme called NNMT (Nicotinamide N-methyltransferase)

Ferroptosis is a unique form of regulated cell death dependent on the build-up of lipid peroxides which consists of lipid peroxidation process [64, 65]

Collectively, these findings support ferroptosis as a therapeutic target to prevent renal fibrosis in CKD