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The net potential of these molecular events is posited to be an environment more favorable to cell survival, in which proliferative signals outweigh apoptotic triggers

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For the first time, it has been demonstrated that the above-mentioned MVs could cause upregulation of collagen I1 and collagen III mRNA expression while having no adverse impacts on the survival and proliferation of both MSCs and fibroblasts

Under high glucose conditions, endothelial cells can acquire the characteristics of fibroblasts, via endothelial-mesenchymal transition (EndMT), which can contribute to neointimal hyperplasia [47]