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Beyond HMGB1, ferroptosis cells trigger inflammatory responses and promote macrophage recruitment by activating molecular pathways that induce inflammation

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Data are normalized to the NAD + abundance

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KYN levels) and additional characterization of expression levels in sensitive and resistant tumor types is warranted among all patient populations

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H19 promote calcium oxalate nephrocalcinosis-induced renal tubular epithelial cell injury via a ceRNA pathway

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