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Those of us in the hypermobility and fibromyalgia space are well aware of the complex mast cell and inflammatory picture in fibromyalgia, and its worth keeping in mind that the anti-inflammatory effects of GLP-1 drugs have been studied in a pretty specific metabolic context, not in the kind of neuroinflammatory picture we see in central sensitisation states
Side effects may be more frequent with tirzepatide
These combined effects lead to improved glycaemic control and significant weight reduction in clinical trials
But I do think there are so many important and well described neuronal populations that have the GLP-1 receptor and which are accessible from the periphery
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