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"Hepatocyte-specific Gclc deletion leads to rapid onset of steatosis with mitochondrial injury and liver failure"

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Small-molecule activators of ATF6 are being developed to enhance ER folding capacity, stimulate ERAD, and reduce ER stress, with potential benefits in heart failure and ischemic cardiomyopathy

Cardiovascular Toxicology, 22 (2), 141151

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