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Interaction between mitochondrial dysfunction and oxidative stress in vitiligo Mitochondrial dysfunction is considered essential in losing redox homeostasis (high spontaneous ROS production and lack of antioxidant network) in vitiligo ( + T cells are significant for the induction of melanocyte apoptosis

Furthermore, PS1V97L transgenic mice exhibited increased inflammatory cytokines including IL-1 and TNF, which were attenuated by sulforaphane supplementation (Hou et al., 2018)

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