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6e, all three GSSG-enhancing chemicals increased the levels of IKK--SSG adducts, suggesting that GSSG accumulation restrained IKK- activity by promoting IKK- S -glutathionylation, leading to subsequent NF-B suppression

At the same time, we also examined the expression of apoptosis-related markers including Caspase-3, Cleaved Caspase-3, Caspase-8, FADD, and GSDME proteins (Fig