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The fact that mutations in the hereditary hemochromatosis protein (HFE) have not been found to increase the risk of AML (89, 90) may also indicate that de novo AML development is not induced by systemic iron overload

Additionally, mast cells directly respond to gliadin fragments (part of the gluten protein) and release proinflammatory mediators.[ref] Related article: Celiac Genes Ehlers-Danlos, Hypermobility, and Mast Cell Activation: As mentioned above, mature mast cells are found in tissues and congregate in the connective tissue around lymphatic vessels, blood vessels, nerves, and skin