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Description

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Conclusion Based on these investigations with a model of normal small airway epithelia and other normal lung epithelial cells, we predict that increased TNF/NFB- and ENaC- dependent inflammation in the COVID-19 airway is due to inhibition of CFTR signaling by SARS-CoV-2 spike protein, thus inducing a pro-inflammatory clinical phenotype in lung epithelia

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Once reconstituted, store at 2-8C and use within 4-6 weeks

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[DOI] [PMC free article] [PubMed] [Google Scholar] 13.Bhatia D., Choi M.E

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