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This results in the formation of disulfide bridges (-S-S-), which in turn leads to decreased hemoglobin solubility and ultimately the irreversible precipitation of oxidized hemoglobin

31,32 The recognition that modification of LDL is an initiator of endothelial damage allows for a clearer understanding of LDLs role in the grand scheme of heart disease

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Dihexa binds to HGF with high affinity and potentiates HGF's ability to activate c-Met at concentrations far below normal thresholds