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In this case, persistent HPA-axis activation, together with PD-1mediated lowering of immune tolerance and postoperative inflammation, may have created a second-hit state that further reduced the threshold for immune activation and facilitated moxifloxacin-induced T-cell cytotoxicity

doi:10.1016/j.heliyon.2017.e00251

History of pancreatitis : GLP-1 agonists may increase the risk of pancreatitis in some individuals

391 GLP-1 and GLP-1 analogs exert their effects on food intake and body weight through a myriad of neural substrates, encompassing several hypothalamic nuclei (including the arcuate nucleus of the hypothalamus, periventricular hypothalamus, and lateral hypothalamic area), hindbrain nuclei (such as the parabrachial nucleus and medial nucleus tractus solitarius), the ventral subregion of the hippocampus (vHP), and nuclei embedded within the mesolimbic reward circuitry (including the ventral tegmental area, VTA, and nucleus accumbens, NAc)