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If these intermediate metabolites are not quickly processed, they can cause massive cellular damage within the liver itself

It was demonstrated that Tax, a HTLV-1-viral protein, induces an increase in ROS generation, causing DNA damage and cellular senescence [17]

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and terminal stage membrane rupture massively discharges immunosuppressive factors like TGF- and adenosine while inducing ferroptosis in T/Natural Killer Cells (NK cells), thereby counteracting antitumor immunity (76)