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After an injury, Thymosin Beta 4 is released by platelets and numerous other types of cells to protection, regeneration, and remodeling of injured/damaged tissues protect the most damaged cells and tissues and to reduce inflammation

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Research suggests it acts through modulation of several interconnected signaling pathways simultaneously a broad mechanism of action that helps explain its observed multisystemic effects in laboratory models, and that also makes it challenging to evaluate through conventional single-target drug development frameworks