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In addition, increasing plasma levels of Apo C-III, which inhibits the activity of LPL in individuals with diabetes, can reduce the catabolism of VLDL.[] Additionally, increasing the transfer of fatty acids to the liver, a common disorder in insulin-resistant diabetes, may increase the secretion of VLDL,[ , ] and also its concentration may rise by higher amounts of fatty acids and Apo B100.[] By stimulating and contributing to the decomposition of fatty acids in the mitochondria, L-carnitine can probably reduce the number of fatty acids to produce Lp (a) in patients who are likely to have high atherogenic lipoprotein production
Investigating surrogate cerebrospinal fluid matrix compositions for use in quantitative lc-ms analysis of therapeutic antibodies in the cerebrospinal fluid
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Its ability to promote wound healing and angiogenesis significantly exceeds that of standard angiogenic growth factors
