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It acts as a physiological ligand for Fe 2 + , promoting assembly of iron-sulfur (Fe-S) clusters and biosynthesis of Fe-S proteins, which participate in essential biological functions including electron transfer, the Krebs cycle, maintenance of genome integrity, enzymatic catalysis, and iron homeostasis 15

If we could hit that downstream process directly, then potentially we wouldnt have to use GLP-1 drugs with their side effects

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Diabetes Care 2020;43:2066-73