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Key Research Findings GHK-Cu has been shown to increase collagen synthesis in fibroblast cultures by stimulating TGF- signaling pathways and upregulating collagen I/III gene expression

These findings are mechanistically coherent with GHK-Cus anti-inflammatory NF-kB activity because the secondary injury cascade in SCI involves microglial activation, sustained pro-inflammatory cytokine release, and reactive oxygen species generation

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