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Furthermore, mitochondrial dysfunction is closely implicated in AKI pathogenesis through the activation of inflammatory signaling cascades, which may lead to PTC apoptosis and necrosis (Jiang et al., 2020

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As detected by JC-1 staining combined with flow cytometry, the PLB-treated group showed a significant enhancement of MMP, reflecting an improvement in mitochondrial function, but YTHDF1 overexpression significantly reduced MMP (Fig

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