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However, prolonged exogenous IGF-1 suppressed the p53/progerin pathway to diminish hepatocyte premature senescence through activating the PI3K/AKT1 pathway (Supplementary Fig

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GHK-Cu: Skin, Tissue, and Nervous System Repair GHK-Cu (Copper Peptide) enhances wound healing, collagen production, and cellular detoxification

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Further research is needed to optimize treatment protocols, develop more personalized approaches, and explore novel combination therapies

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