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In general, most hydrophobic BAs are considered as FXR agonists, while hydrophilic non-12-OH TMCA, UDCA, and T(G)UDCA serve as endogenous FXR antagonists 4,9,10

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The study concluded that probiotics reduced gut leakiness, lowered oxidative stress, and enhanced GABA-receptor expression, supporting a protective role on the gut-brain axis in this ASD model