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The core pathophysiological alteration in PMOS involves dysfunction of the hypothalamic-pituitary-ovarian axis, primarily characterized by dysregulated GnRH release and excessive pulsatile secretion of LH

One of the theories that tries to explain the exercise effects defends that it may be in part due to the accumulation of oxidative damage induced by reactive oxygen species (ROS) and reactive nitrogen species (RNS) to cells and macromolecules [13]

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Bastenie PA, Vanhaelst L, Golstein J, Smets P, Keys MJ, Karvonen MJ, Punsar S